Elbow Dislocation

Elbow

Overview

Elbow dislocation is the second most common major joint dislocation in adults (after the shoulder) and the most common major joint dislocation in children. Simple (non-complex) dislocations with no associated fracture account for 50-60% of cases and have a generally favourable prognosis with prompt closed reduction and early mobilisation. Complex dislocations (with associated fracture) carry a higher risk of instability, stiffness, and post-traumatic arthritis, and usually require operative management. Posterolateral is by far the most common pattern. The elbow is intrinsically bony-stable; for dislocation to occur, significant ligamentous disruption is always present, even in cases that reduce easily. BOAST-aligned open-injury principles apply to open dislocations.

Anatomy & Pathophysiology

The elbow comprises three articulations within a single synovial capsule: ulnohumeral (hinge), radiocapitellar (rotation), and proximal radioulnar (pivot). Bony congruence from the trochlea-olecranon articulation and the radial head buttressing the capitellum makes the elbow intrinsically stable; dislocation therefore requires substantial soft-tissue disruption.

Key stabilisers:

  • Medial ulnar collateral ligament (MUCL) complex: anterior, posterior, and transverse bundles. The anterior bundle is the primary restraint to valgus stress between approximately 20-120 degrees of flexion
  • Lateral collateral ligament (LCL) complex: radial collateral, annular, accessory lateral collateral, and the lateral ulnar collateral ligament (LUCL). The LUCL is the primary restraint to posterolateral rotatory instability (PLRI)
  • Radial head: secondary restraint to valgus stress and a key stabiliser against posterior subluxation, particularly when the MUCL is deficient
  • Coronoid process: the keystone anterior buttress preventing posterior dislocation. Even small coronoid fractures destabilise the joint
  • Joint capsule and the flexor-pronator and extensor origins: dynamic and secondary static stabilisers

Horii circle (three-stage lateral-to-medial soft-tissue failure, per O'Driscoll):

  • Stage 1: LUCL disruption (posterolateral rotatory instability)
  • Stage 2: anterior and posterior capsule disruption (perched dislocation)
  • Stage 3: medial-sided failure - 3A posterior MCL bundle disruption (reducible but rotationally unstable); 3B complete MCL disruption (frank dislocation, reducible but very unstable); 3C stripping of the flexor-pronator origin (extreme instability, often operative)

Neurovascular structures at risk: median nerve and brachial artery anteriorly; ulnar nerve behind the medial epicondyle (vulnerable in medial-sided injury, medial epicondyle avulsions, and later traction during contracture rehabilitation); radial nerve and PIN rarely injured in pure dislocation but vulnerable in fracture-dislocations involving the proximal radius.

Terrible triad of the elbow: posterior dislocation plus radial head fracture plus coronoid fracture. High instability, high complication rate, almost always operative.

Classification by direction: posterior or posterolateral (the dominant pattern, more than 80%); posteromedial (varus stress pattern, associated with coronoid anteromedial facet fracture); anterior, medial, lateral, or divergent are uncommon.

Mechanism: typically a fall on outstretched hand with the elbow extended, forearm supinated, and a valgus load producing posterolateral dislocation. High-energy mechanisms include motor vehicle collisions and contact sports.

Clinical Pearl

The elbow's intrinsic bony stability means a simple dislocation that reduces easily is NOT a benign soft-tissue injury - significant capsuloligamentous disruption is always present. The Horii circle (three-stage lateral-to-medial failure with 3A-3C subdivisions) explains why early specialist follow-up and structured rehabilitation matter even after straightforward reduction.

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