Lateral epicondylalgia (commonly called tennis elbow, more formally lateral elbow tendinopathy) is a tendinopathy of the common extensor origin at the lateral epicondyle, most often involving extensor carpi radialis brevis (ECRB). Peak incidence age 35-55, men and women equally affected. Despite the name, under 10% of cases relate to racquet sports - most occur in occupational settings involving repetitive gripping, wrist extension, and forearm rotation. Prevalence 1-3%. The condition is usually self-limiting, with most patients recovering within around 1 year.
The common extensor origin arises from the lateral epicondyle and includes ECRB, extensor digitorum, extensor carpi ulnaris, and extensor digiti minimi. ECRB is most commonly affected because it lies deepest against the capitellum and is subject to compressive and shear forces during gripping and wrist extension.
The pathology is tendinopathy, not tendinitis: angiofibroblastic degeneration with disorganised collagen, neovascularisation, increased ground substance, and absence of inflammatory cells. This is a failed healing response with a neurogenic pain component. The outdated inflammatory model has direct management consequences: anti-inflammatory drugs and corticosteroid injection do not address the underlying pathology.
Cook and Purdam tendon continuum:
Risk factors: repetitive gripping and wrist extension tasks, age 35-55, smoking, obesity, diabetes, and coexisting tendinopathy elsewhere (tendinopathic diathesis).
ECRB is the key tendon - deepest at the common extensor origin and compressed against the capitellum during gripping. The pathology is angiofibroblastic degeneration, not inflammation, which is why corticosteroid injection produces short-term relief but worse long-term outcomes (Coombes 2013).
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