TMJ Injury and Dysfunction

Head & Face

Overview

The temporomandibular joint (TMJ) is one of the most-used joints in the body and one of the most-overlooked in MSK and SEM teaching. Pathology spans an acute traumatic spectrum (subluxation, dislocation, condylar fracture) and a chronic pain-and-dysfunction spectrum collectively termed temporomandibular disorders (TMD). The SEM clinician most often encounters post-traumatic dysfunction after blunt facial trauma, acute dislocation pitchside or in ED, and primary TMD with jaw pain, clicking, and headache. Key decisions: recognise acute injuries that need same-day intervention (anterior dislocation requiring reduction; condylar fracture requiring urgent OMFS pathway), distinguish primary TMD (multimodal conservative care) from sinister mimics (giant cell arteritis is the most important), and know when SEM scope ends and OMFS, restorative dentistry, or specialist orofacial pain services take over. UK practice anchors on the 2025 RCS/FDS guideline, NICE CKS, and NICE NG232 for any associated head injury.

Anatomy & Pathophysiology

The TMJ is a paired synovial joint between the mandibular condyle and the glenoid fossa of the temporal bone, separated by a fibrocartilaginous articular disc that divides the joint into superior and inferior compartments. The disc is anchored anteriorly to the lateral pterygoid muscle, which drives protrusion and contralateral deviation. Movement combines hinge (rotation in the inferior compartment, opening up to ~25 mm) and translation (condyle and disc gliding forward over the articular eminence in the superior compartment, additional opening to a normal maximal unassisted inter-incisal distance of at least 35 mm).

Four named pathology patterns:

  • Acute dislocation: most commonly anterior (mouth locked open, palpable preauricular hollow). Less commonly posterior, lateral, or superior - these usually accompany skull base injury
  • Condylar fracture: intracapsular, condylar neck, or subcondylar. The condylar neck acts as a protective fuse, sparing the middle cranial fossa
  • Primary TMD: an umbrella covering myofascial pain, internal derangement (disc displacement with or without reduction), and inflammatory or degenerative arthralgia
  • Post-traumatic TMD: dysfunction after blunt facial trauma, whiplash, or missed condylar fracture

Triggers for anterior dislocation include excessive yawning, dental procedures, vomiting, seizures, and direct trauma to the chin. Recurrent dislocation is common after a first episode because of capsular laxity.

Clinical Pearl

Anterior dislocation: the condyle slips forward over the articular eminence and gets stuck. Mouth locked open with palpable preauricular hollow. Reduce with GAUZE-WRAPPED thumbs on the lower molars and slow inferoposterior pressure - downward first to disengage, then posterior to walk back into the fossa. Gauze protects against the reflex bite.

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