Cervical Stinger / Brachial Plexus Neuropraxia

Spine

Overview

A cervical stinger or burner is a transient, unilateral upper-trunk brachial plexus or cervical nerve root neuropraxia, classically experienced after a contact-sport collision. The player walks off the pitch shaking their arm, with burning electric pain shooting from neck into arm and transient weakness or paraesthesia, usually resolving within seconds to minutes. It is common in rugby, American football, ice hockey, and wrestling. The clinical priority is to distinguish a benign unilateral stinger from sinister mimics - cervical spine fracture, cord neuropraxia (bilateral or four-limb symptoms), concomitant concussion, and true cervical radiculopathy - which trigger NICE NG41 and BOASt pathways.

Anatomy & Pathophysiology

The brachial plexus is formed from the ventral rami of C5 to T1, passing through the scalene triangle and under the clavicle to supply the upper limb. The upper trunk (C5-C6) is most commonly affected, which explains the typical sensory and motor distribution: shoulder, lateral arm, deltoid, biceps, and supraspinatus / infraspinatus.

Three mechanisms produce a stinger:

  • Traction: lateral neck flexion away from the affected side with simultaneous shoulder depression stretches the upper trunk - the classic young-athlete or first-time pattern
  • Compression: forced ipsilateral lateral flexion plus extension compresses the exiting nerve root in the neuroforamen - dominates in older athletes with degenerative foraminal narrowing
  • Direct blow to Erb's point in the supraclavicular fossa compresses the upper trunk directly
Stinger mechanism: lateral neck flexion plus shoulder depression stretches the upper trunk (C5-C6) of the brachial plexus, producing unilateral transient burning pain.

The classic transient stinger is a Sunderland grade I neuropraxia (focal demyelination, axonal continuity preserved). Prolonged or recurrent stingers may include grade II axonotmesis with longer recovery and persistent weakness.

Clinical Pearl

Three mechanisms, three patterns. Traction = lateral flexion AWAY + shoulder depression (young athletes). Compression = lateral flexion TOWARDS + extension (older, foraminal narrowing). Direct blow to Erb's point. All produce upper-trunk C5/C6 symptoms.

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