Plantar fasciitis - increasingly termed plantar fasciopathy or plantar heel pain - is the most common cause of inferior heel pain in adults. It results from repetitive overload at the plantar fascia origin on the medial calcaneal tuberosity, with degenerative tissue change rather than active inflammation, hence the shift toward fasciopathy. It typically affects adults aged 40-60, with higher prevalence in women, runners, and patients with obesity. The cardinal symptom is first-step pain. Approximately 80-90% resolve within 12 months of conservative management, though recovery can be prolonged.
The plantar fascia originates from the medial calcaneal tuberosity and fans distally into five slips inserting at the proximal phalanges. It is the primary static stabiliser of the medial longitudinal arch.
The windlass mechanism (Hicks): during toe-off, dorsiflexion of the toes tightens the fascia around the metatarsal heads, rigidifying the arch for push-off. Cyclical loading drives pathology at the medial calcaneal insertion.
Histology shows a degenerative process (fasciosis) rather than active inflammation: collagen disorganisation, neovascularisation, and absence of inflammatory cells - analogous to tendinopathy.
Heel spurs are traction enthesophytes seen in around 50% of cases but in 20% of asymptomatic individuals. They are incidental and not the cause of pain. Spur excision is not indicated.
Major modifiable risk factors: obesity, prolonged standing, sudden training increase, and reduced ankle dorsiflexion from calf tightness.
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