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Achilles tendinopathy is a common overuse condition affecting the Achilles tendon, the largest and strongest tendon in the human body. It is characterised by pain, swelling, and impaired function, most frequently seen in running and jumping athletes but also prevalent in the general population. The condition can be classified as mid-portion (2-6 cm above the calcaneal insertion) or insertional, with distinct pathological and management considerations for each.
The Achilles tendon is formed from the confluence of the gastrocnemius and soleus muscles and inserts onto the posterior calcaneus. It transmits forces of up to 12.5 times body weight during running. The mid-portion of the tendon has a relatively hypovascular zone, thought to contribute to its vulnerability to degeneration.
The current understanding of tendinopathy has moved away from an inflammatory model toward one of failed healing. The continuum model (Cook & Purdam) describes three stages: reactive tendinopathy, tendon dysrepair, and degenerative tendinopathy. Histologically, there is an increase in ground substance, collagen disorganisation, and neovascularisation without true inflammation.
Remember the tendon continuum as 'React → Repair → Degenerate'. Reactive tendinopathy is reversible with load management; degenerative tendinopathy is not. Treatment strategy depends on where the tendon sits on this continuum.
Patients typically report gradual onset of pain localised to the Achilles tendon, often described as stiffness in the morning or after periods of inactivity. Pain is commonly aggravated by increased loading activities such as running, jumping, and hill walking, and may improve with gentle activity (the so-called 'warm-up' phenomenon).
On examination, there may be a palpable thickening or nodule in the mid-portion of the tendon. The Royal London Hospital test (pain on palpation that decreases with the tendon under tension) may be positive. Insertional tendinopathy presents with pain at the calcaneal attachment, often with a prominent posterior heel. Single-leg heel raises may reproduce symptoms and assess tendon capacity.
Achilles tendinopathy is primarily a clinical diagnosis. However, imaging can be helpful to confirm diagnosis, assess severity, and exclude differential diagnoses.
Ultrasound is the first-line imaging modality and can demonstrate tendon thickening, hypoechoic areas, loss of fibrillar pattern, and neovascularisation on Doppler. MRI provides excellent soft tissue contrast and is useful for assessing intrasubstance tears, paratenon involvement, and insertional pathology including retrocalcaneal bursitis and Haglund's deformity.
Plain radiographs may show calcification at the insertion in chronic insertional tendinopathy. Blood tests are not routinely indicated unless systemic inflammatory or metabolic conditions are suspected.
Management of Achilles tendinopathy is predominantly conservative, with strong evidence supporting exercise-based rehabilitation as the cornerstone of treatment.
First-line management includes load management advice, activity modification, and a structured exercise programme. Eccentric exercises (the Alfredson protocol) have historically been the most studied, but recent evidence supports heavy slow resistance training and isometric loading for pain relief as equally effective alternatives.
Adjunctive treatments include shockwave therapy (ESWT), which has moderate evidence for mid-portion tendinopathy. GTN patches and peritendinous injections (high-volume image-guided injection) may be considered in refractory cases. Corticosteroid injections should be used with caution due to the risk of tendon rupture.
Surgical intervention is reserved for cases that fail 6-12 months of appropriate conservative management and may include open or minimally invasive procedures such as tendon debridement, ventral paratenon stripping, or Haglund's resection for insertional disease.
Rehabilitation follows a progressive loading approach, typically divided into four stages:
1. Isometric loading: Used in the acute reactive phase for pain relief and to maintain tendon capacity. Typically 5 × 45-second holds at 70% MVC.
2. Isotonic strengthening: Heavy slow resistance training (HSR) with exercises such as seated and standing calf raises, progressing load over 12 weeks. The Alfredson eccentric protocol (3 × 15 reps, twice daily) remains a well-supported alternative.
3. Energy storage and plyometrics: Introduction of hopping, skipping, and bounding activities to restore the tendon's capacity for energy storage and release.
4. Return to sport: Sport-specific drills, graduated return to full training. Criteria include pain-free single-leg heel raises × 20, symmetrical calf strength, and completion of sport-specific loading without symptom flare.
Timeline: Most patients require 3-6 months of rehabilitation. Athletes should be counselled that full recovery may take 6-12 months.
Insertional Achilles tendinopathy (IAT) requires a modified rehabilitation approach. Eccentric and heavy slow resistance loading for IAT should be performed on a flat surface rather than dropping below horizontal off a step - deep ankle dorsiflexion compresses the insertion against the posterior calcaneus, worsening the compressive component of the pathology.
A small heel lift (6-12 mm) is commonly used during symptomatic periods to reduce insertional compression and is particularly useful if there is coexisting Haglund deformity. This is distinct from mid-portion tendinopathy where full dorsiflexion loading off a step is well tolerated and part of the standard Alfredson protocol.
NICE CKS: Achilles tendinopathy - primary care framework with referral thresholds
cks.nice.org.uk
BOFAS (British Orthopaedic Foot and Ankle Society) - patient resources and consensus statements
bofas.org.uk
BOA Standards for Trauma (BOASTs) - UK trauma standards
boa.ac.uk
BNF: NSAIDs treatment summary
bnf.nice.org.uk