Shoulder Instability

Shoulder

Overview

Shoulder instability spans a spectrum from subluxation to frank dislocation, with the humeral head translating abnormally relative to the glenoid. Anterior instability accounts for approximately 95% of traumatic dislocations. Peak incidence is in males aged 15-30 (contact sport, trauma) with a second peak in older adults (low-energy falls). Recurrence is strongly age- and sex-dependent and is highest in younger males - most recurrent dislocations occur within 2 years of the first event.

Anatomy & Pathophysiology

The glenohumeral joint sacrifices bony congruity for mobility. Static stabilisers: glenoid labrum (deepens the socket by up to 50%), the glenohumeral ligaments (the inferior glenohumeral ligament complex, IGHLC, is the primary restraint to anterior translation in abduction and external rotation), and the joint capsule. Dynamic stabilisers: rotator cuff and scapular stabilisers.

Classification - TUBS / AMBRI:

  • TUBS: Traumatic, Unidirectional, Bankart lesion, Surgery typically required
  • AMBRI: Atraumatic, Multidirectional, Bilateral, Rehabilitation, Inferior capsular shift if surgery considered

Stanmore Polar Triangle (contemporary UK model):

  • Type I: Traumatic structural - discrete pathological lesion (Bankart, bony Bankart, Hill-Sachs)
  • Type II: Atraumatic structural - capsular laxity, often with generalised hypermobility
  • Type III: Muscle patterning / non-structural - abnormal muscle firing patterns cause habitual instability without structural lesion. These patients need specialist physiotherapy, NOT surgery. Operating on Type III is a recognised pitfall.

Traumatic anterior - key pathological lesions:

  • Bankart lesion: detachment of the anteroinferior labrum from the glenoid rim - the essential lesion of traumatic anterior instability. Variants include bony Bankart (with glenoid rim fracture, changing surgical approach) and ALPSA (medialised healing).
  • Hill-Sachs lesion: compression fracture of the posterosuperior humeral head; engaging Hill-Sachs lesions contribute to recurrent instability
  • HAGL (humeral avulsion of the glenohumeral ligament): hidden cause of recurrent instability that may be missed if only labral pathology is assessed
  • Concomitant rotator cuff tear: common in patients over 40 after first-time anterior dislocation - cuff integrity must be assessed
The bipolar lesion pattern after anterior glenohumeral dislocation. Anteroinferior labral disruption (Bankart) and posterosuperolateral humeral head impaction (Hill-Sachs) commonly occur together. Engagement of a Hill-Sachs defect with the anterior glenoid rim during external rotation in abduction drives recurrent instability.

Atraumatic instability: generalised ligamentous laxity (Beighton score), capsular redundancy without discrete labral tear. Common in young females and overhead athletes.

Posterior instability: uncommon (<5%). Classically follows seizure, electrocution, or high-energy trauma. Lightbulb sign on AP X-ray is a classic but subtle clue.

Recurrence risk factors: young age at first dislocation (strongest), male sex, contact sport, significant bony lesion, hyperlaxity.

Clinical Pearl
  • The Stanmore Polar Triangle adds Type III (muscle patterning) to the older TUBS / AMBRI framework - these patients need specialist physiotherapy, NOT surgery
  • Operating on a Type III patient is a recognised pitfall and a commonly tested examiner trap
  • Age at first dislocation remains the strongest predictor of recurrence

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