Mallet finger is disruption of the terminal extensor tendon mechanism at the distal interphalangeal (DIP) joint, producing a characteristic flexion deformity with loss of active DIP extension. It is the commonest closed extensor tendon injury at the hand. The classic mechanism is a sudden forced flexion of an extended DIP - a "jammed" finger when a ball strikes the fingertip during sport, or stubbing the finger while bed-making or catching a falling object. Most cases are treated conservatively with continuous DIP extension splinting for 6-8 weeks, with excellent outcomes when splint compliance is good. Operative fixation is reserved for displaced bony mallet with significant articular involvement or volar subluxation of the distal phalanx.
The terminal extensor tendon is the conjoined insertion of the extensor digitorum communis (with extensor indicis and digiti minimi contributing to their respective digits), the lateral bands, and the triangular ligament, attaching to the dorsal base of the distal phalanx. Active DIP extension requires an intact terminal tendon under appropriate tension.
Mallet injury is either:
Doyle classification: Type I closed tendinous; Type II open tendinous (laceration); Type III open with skin loss; Type IV bony, with subtypes IVA (physeal in children), IVB (fragment under 50% articular surface without subluxation), and IVC (over 50% articular surface or with subluxation). Type IVC is the operative-trigger subtype.
Uncorrected mallet leads to a chronic mallet posture with secondary swan-neck deformity (compensatory PIP hyperextension as extensor force redirects proximally), particularly in patients with lax volar plates.
Doyle IVC (bony mallet with over 50% articular surface or volar subluxation of the distal phalanx) is the operative indication. Sub-50% non-displaced bony mallets can usually be managed in extension splinting alongside soft-tissue mallets.
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