Frozen Shoulder

Shoulder

Overview

Frozen shoulder (adhesive capsulitis) is a self-limiting condition characterised by progressive painful restriction of both active and passive glenohumeral movement. Symptoms typically run for 2-5 years; complete resolution is common but not universal, with up to 40% of patients retaining some residual stiffness. Peak incidence age 40-60, more common in women. Prevalence 2-5% in the general population, rising to 10-38% in diabetes mellitus. Classified as primary (idiopathic) or secondary (post-trauma, post-surgical, or systemic disease).

Anatomy & Pathophysiology

The glenohumeral joint capsule is a synovial-lined structure with recesses (notably the axillary recess and rotator interval) that allow full range of movement. In frozen shoulder, fibroblastic proliferation and collagen deposition cause capsular thickening and contracture, particularly of the rotator interval, coracohumeral ligament, and axillary recess. The capsule loses its normal elastic recess capacity, mechanically restricting glenohumeral motion across all planes.

Risk factors: diabetes mellitus is the strongest association (up to 5x risk and particularly bilateral or treatment-resistant disease), thyroid disease, Dupuytren's, cardiac disease, previous shoulder surgery or prolonged immobilisation. Bilateral involvement occurs in up to 40-50% of diabetic patients.

Clinical Pearl
  • Diabetes is the strongest risk factor, with up to 5x prevalence and a particular association with bilateral and treatment-resistant disease
  • Consider HbA1c testing in any new presentation where diabetes status is unknown
  • The axillary recess and rotator interval are the principal sites of capsular thickening - this anatomical specificity drives the capsular pattern of restriction

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